EKG

Narrow Complex Tachycardia

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Narrow Complex Tachycardia, Supraventricular Tachycardia, Paroxysmal Supraventricular Tachycardia, PSVT

  • See Also
  1. Atrioventricular Nodal Reentry (AVNRT)
  2. Atrioventricular Reciprocating Tachycardia (AVRT)
  3. Atrial Tachycardia
  4. Unstable Tachycardia
  5. Sinus Tachycardia
  6. Atrial Fibrillation
  7. Wide Complex Tachycardia
  8. Cardiopulmonary Resuscitation
  • Definitions
  1. Supraventricular Tachycardia
    1. Rapid heart rhythms originating in the atrium or Atrioventricular Node
    2. Transmit via the Bundle of His and result in rapid ventricular response
  • Epidemiology
  1. Prevalence: 2-3 per 1000 persons (U.S.)
  2. Age (mean): 45 years old
  3. Gender: Women represent 62% of cases
  • Causes
  • Narrow Complex Tachycardia (Mnemonic: 5H 5T PS)
  1. Similar list to Reversible Causes of Cardiopulmonary Arrest (5H5T)
  2. Hypoxemia
  3. Hypovolemia
  4. Hyperthermia
  5. Hyperkalemia
  6. Hypokalemia
  7. Tamponade (Cardiac Tamponade)
  8. Tension Pneumothorax
  9. Toxins, medications and drugs
    1. Caffeine
    2. Tobacco
    3. Alcohol
    4. Pseudophedrine (or other Sympathomimetics)
  10. Thrombus
    1. Myocardial Infarction
    2. Pulmonary Embolism
  11. Pain or Stress
  12. Structural abnormalities (especially in children)
    1. Hypertrophic Cardiomyopathy
    2. Congenital Heart Disease
  • Types
  • Narrow Complex Tachycardia
  1. Sinus Tachycardia
  2. Supraventricular Tachycardia
    1. Atrioventricular Nodal Reentry (AVNRT)
      1. Signal down the slow AV nodal pathway and retrograde up the fast AV nodal pathway
      2. In 10% of cases, the signal reentry route is reversed
    2. Atrioventricular Reciprocating Tachycardia (AVRT)
      1. Includes Wolff-Parkinson-White Syndrome (characterized by delta wave)
      2. Accessory pathway outside the AV Node
        1. Orthodromic (narrow complex): Signal down the AV Node and up the accessory path
        2. Antidromic (wide complex): Signal down the accessory path and up the AV Node
    3. Atrial Tachycardia (AT)
      1. Abnormal focus of atrial automaticity (outside the SA Node)
      2. Unlike AVNRT and AVRT, no accessory pathway is involved
    4. Junctional Ectopic Tachycardia
  3. Other Atrial Tachycardias with rapid ventricular response
    1. Atrial Fibrillation
    2. Atrial Flutter
  • Differential Diagnosis
  • Adults - Narrow Complex Tachycardia (key question is 'regular or irregular')
  1. Sinus Tachycardia (regular)
  2. Irregular Supraventricular Tachycardia (may also present as wide complex if aberrancy)
    1. Atrial Fibrillation
  3. Regular Supraventricular Tachycardia
    1. Atrial Flutter
    2. Atrioventricular Nodal Reentry (AVNRT): 60% of SVT cases (esp. women)
    3. Atrioventricular Reciprocating Tachycardia (AVRT): 30% of SVT cases
    4. Atrial Tachycardia (AT): 10% of SVT cases
    5. Junctional Ectopic Tachycardia
    6. Secondary causes (very high Heart Rates >220-240, refractory or recurrent tachydysrythmia)
      1. Catecholamine surge
      2. Sympathomimetic Toxicity
      3. Hyperthyroidism (Thyrotoxicosis, Thyroid Storm)
  • Differential Diagnosis
  • Children - Narrow Complex Tachycardia
  1. Common
    1. Orthodromic Atrioventricular Reciprocating Tachycardia (Orthodromic AVRT, or ORT)
      1. Most common in children (typical, narrow complex SVT)
    2. Atrioventricular Nodal Reentry (AVNRT)
      1. Second most common SVT in children (but rare in young children)
  2. Uncommon
    1. Ectopic Atrial Tachycardia
      1. Similar to sinus rhythm except for altered P Wave appearance and more rapid rate
    2. Atrial Flutter (uncommon in children outside the newborn period)
    3. Atrial Fibrillation (very rare in children)
  3. Congenital Heart Disease history
    1. CHD predisposes patients to scarring with risk of reentrant pathways
    2. Intra-atrial reentrant Tachycardia (IART)
      1. Appears similar to Atrial Flutter
      2. Typically treated with rate control on Diltiazem (if over age 2-5 years), followed by cardioversion
  • History
  1. Timing
    1. Rapid onset and resolution
      1. Supraventricular Tachycardia
    2. Slow onset and resolution
      1. Sinus Tachycardia
  2. Precipitating factors
    1. Caffeine or stimulants, stress
      1. Supraventricular Tachycardia or Sinus Tachycardia
    2. Cardiovascular disease or onset with activity
      1. Ventricular Tachycardia
  • Symptoms
  • Episodic
  1. Anxiety
  2. Chest pressure
  3. Dyspnea
  4. Fatigue
  5. Light headed
  6. Palpitations
  1. Sinus Tachycardia
    1. P Waves present and normal
      1. At higher rates P Wave may be hidden in T Wave
      2. Look for high frequency notching within the T Wave to suggest a hidden P Wave
    2. Variable R-R with constant PR Interval
    3. Heart Rate varies with activity
    4. Rate lower than PSVT
      1. Infants < 220 (may approach this with fever)
      2. Children < 180
      3. Adults < 160
  2. Supraventricular Tachycardia
    1. P Waves absent or abnormal
    2. Fixed Heart Rate (constant R-R)
    3. Abrupt rate change
    4. Rate higher than Sinus Tachycardia (especially if Heart Rate twice normal for age or higher)
      1. Infants > 220
      2. Children > 180
      3. Adults > 160
        1. Very high rates >220-240 suggests underlying secondary cause (e.g. Thyrotoxicosis, see above)
  • Labs
  1. Precautions
    1. Paroxysmal Supraventricular Tachycardia does not require routine labs in many cases (esp. known prior history of PSVT)
    2. Patients who are asymptomatic after PSVT resolves, and without underlying other risks need not undergo laboratory testing
  2. Initial labs to consider
    1. Thyroid Stimulating Hormone (TSH)
    2. Basic metabolic panel
    3. Complete Blood Count
    4. Chest XRay
  3. Additional tests to consider (in adults)
    1. Holter Monitor or Event Monitor
    2. Thyroid Stimulating Hormone (TSH)
  4. Additional tests to consider for concerns of underlying Cardiomyopathy
    1. Brain Natriuretic Peptide (ntBNP)
    2. Echocardiogram
  1. Background
    1. No lab testing is indicated in routine PSVT that resolves without symptoms
    2. Repeat EKG and reevaluate patient history and exam 15 minutes after return to sinus rhythm
      1. Asymptomatic patients with normal ekg and exam after return to sinus rhythm need no further testing
      2. Consider lab testing driven by specific positive symptoms, signs or risk factors after return to sinus rhythm
  2. Serum Troponin Is NOT typically indicated in Supraventricular Tachycardia (SVT)
    1. SVT is rarely due to acute occlusive Myocardial Infarction
    2. ST Depression is commonly seen as a stress response to Tachycardia, but is not indicative of a coronary event
    3. Serum Troponin Is often elevated in SVT (esp. if prolonged) but is not associated with 90 day adverse outcomes
  3. Despite low efficacy and resulting Cascades of Care, Troponin Is obtained in 80% of ED SVT evaluations (abnormal in 30% of cases)
    1. Gabrielli (2022) Cardiol Rev +PMID: 35148534 [PubMed]
  4. Troponin Indications
    1. Persistent concerning EKG changes after conversion to sinus rhythm
    2. Symptoms and signs specifically suggestive of underlying Acute Coronary Syndrome (not attributable to SVT alone)
  5. References
    1. Mattu and Swaminathan (2022) EM:Rap 22(12): 7-8
  • Management
  • Stable Patients
  1. New emphasis on use of one Antiarrhythmic
    1. Contrast to prior Antiarrhythmic soups
    2. Pro-arrhythmic effects increase with poly-drugs
  2. Supraventricular Tachycardia Management in the Adult
  3. Supraventricular Tachycardia Management in the Child
  1. See Unstable Tachycardia
  1. Uncertain diagnosis or management
  2. Recurrent Supraventricular Tachycardia refractory to medications
  3. High risk profession or activity (e.g. truck driver, airline pilot, rock climbing, Scuba Diving)
  4. Ablation considered over medication (patient preference)
  5. Wolff-Parkinson-White Syndrome (characterized by delta wave)
  6. Structural heart disease (e.g. Hypertrophic Cardiomyopathy)
  7. Syncope associated with Supraventricular Tachycardia
  8. Wide complex on EKG
  • References
  1. Claudius, Behar and Bar Cohen in Herbert (2014) EM:Rap 14(5):1-2
  2. Hebbar (2002) Am Fam Physician 65(12):2479-86 [PubMed]
  3. Helton (2015) Am Fam Physician 92(9): 793-800 [PubMed]
  4. Cardiopulmonary Resuscitation Guidelines
    1. http://www.circulationaha.org
    2. (2010) Guidelines for CPR and ECC [PubMed]
    3. (2005) Circulation 112(Suppl 112):IV [PubMed]
    4. (2000) Circulation, 102(Suppl I):86-9 [PubMed]