Exam
Newborn Cardiopulmonary Exam
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Newborn Cardiopulmonary Exam
, Newborn Chest Exam
See Also
Newborn Resuscitation
Pediatric Murmur
Congenital Heart Disease
Neonatal Cyanosis
Fetal Tachycardia
Fetal Bradycardia
Physiology
See
Fetal Circulation
Exam
Breast
and Xiphisternum
Palpate for abnormality
Inspection
Unilateral absence or hypoplasia of pectoralis major
Poland's Syndrome
(Poland's Sequence)
Chest
Deformity
Pectus Carinatum
Pectus Excavatum
Widely spaced nipples
Turner's Syndrome
Noonan Syndrome
Breast
Buds and
Galactorrhea
Resolves in first month of life
Consult pediatric endocrinology if persistent
Exam
Respiratory
See
Respiratory Distress in the Newborn
Brief periods apnea are normal in transition
Periodic breathing
Common and normal in children
Pauses in breathing up to 10-20 seconds are normal
Unless associated
Bradycardia
, mental status change, or
Skin Color
change
Apnea >20 seconds is abnormal and should prompt observation
RSV Bronchiolitis
in age 6-8 weeks is a risk for signficant apnea
Evaluate for unequal breath sounds or unequal chest wall movement
Pneumothorax
Diaphragmatic Hernia
Cyst
ic malformation of the lung
Observe for respiratory distress
Tachypnea
, grunting or
Cyanosis
Nasal flaring or intercostal retractions
Consider causes (See
Respiratory Distress in the Newborn
)
Respiratory Distress Syndrome in the Newborn
Meconium Aspiration Syndrome
Transient Tachypnea of the Newborn
Stridor
Laryngomalacia
(most common)
Larynx
region collapses in the first few months of life in some infants (resolves by age 1-2 years)
Better when supine and worse with feeding
Considered benign if mild in an otherwise well appearing infant with normal growth
Other
Stridor
causes to consider
Serious airway Infections (croup,
Bacterial Tracheitis
,
Epiglottitis
)
Subglottic Stenosis
(infants that were previously intubated)
Vocal Cord Paralysis
(prior neck surgery)
Hemangioma
with local neck compression
Exam
Cardiovascular
See
Congenital Heart Disease
Vitals (Normal Newborn)
See
Pediatric Vital Signs
See
Newborn Pulse Oximetry Screening for Congenital Heart Disease
Pulse Oximetry
screening for
Congenital Heart Disease
(prior to discharge at 24 hours)
Appearance
Cyanosis
and
Tachypnea
frequently accompany
Congenital Heart Disease
Abnormal Pulse
s
Diminished pulses in all extremities
Diminished
Cardiac Output
(e.g.
Aortic Stenosis
)
Peripheral
Vasocon
striction
Diminished femoral pulses
Heart lesion dependent on ductus arteriosus (e.g.
Aortic Coarctation
)
Bounding Pulse
s
High
Cardiac Output
(e.g.
Patent Ductus Arteriosus
)
Hypertension
(See normal
Blood Pressure
above)
See
Hypertension in Infants
Murmurs
See
Pediatric Murmur
Pathologic murmur characteristics
Holosystolic murmur,
Diastolic Murmur
or continuous murmur
Harsh
Heart Murmur
or Grade 3 murmur or louder
Newborn murmurs are often transient
Tricuspid regurgitation
Patent Ductus Arteriosus
Altered position of precordial heart sounds
Shifted right:
Dextrocardia
Gallup Rhythms
Split S2
Normal finding
Split S2 is Absent
Common with
Cyanotic Congenital Heart Disease
(
Truncus Arteriosus
,
Hypoplastic Left Heart
, pulmonic valve atresia)
Results in high pulmonary vascular resistance
References
Drapkin (2019) Am J Emerg Med 37(6):1153-9 +PMID:30952605 [PubMed]
Lewis (2014) Am Fam Physician 90(5): 289-96 [PubMed]
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