C-Spine
Acute Calcific Tendonitis of the Longus Colli
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Acute Calcific Tendonitis of the Longus Colli
, Longus Colli Tendonitis, ACTLC
See Also
Neck Pain
Odynophagia
Globus Hystericus
Epidemiology
Ages 30 to 60 years old
No significant gender predisposition
Uncommon: 0.5 cases per 100,000 person years
Pathophysiology
Longus coli is one of 4 cervical flexor
Muscle
s
Calcific
Tendonitis
develops within the longus coli
Muscle
tendons
Superior aspect tendons are most susceptible (C1 to C3, down to C5 region)
Calcium
deposits within tendon as a response to decreased local strength
Tendon rupture results in release of hydroxyapatite crystals into soft tissue
Local crystal release results in aseptic inflammatory response
Precipitating injuries
Repetitive
Trauma
with secondary localized tendon ischemia and necrosis
Course
Resolves spontaneously over weeks
Resolves as hydroxyapatite crystals are phagocytosed by
Macrophage
s
Symptoms
Acute
Neck Pain
Neck stiffness and decreased range of motion
Dysphagia
Odynophagia
Globus Hystericus
Low grade fever
Labs
Leukocytosis
(mild)
Inflammatory markers (e.g. CRP) mildly increased
Imaging
CT
Cervical Spine
Gold standard for identifying specific calcification changes of ACTLC
Calciium deposition within the longus colli
Prevertebral edema
Rim enhancement is absent in ACTLC (but present in abscess)
Differential Diagnosis
See
Neck Pain
See Odynophagia
See
Globus Hystericus
Retropharyngeal Abscess
Meningitis
Spinal Epidural Abscess
Spinal Epidural Hematoma
Cervical Disc Herniation
Vertebra
l
Fracture
Management
Conservative management
Resolves over the course of weeks spontaneously
No indication for extracorporeal shock wave therapy or surgery
Analgesic
s and antiinflammatory agents
NSAID
s
Corticosteroid
s
References
Raggio (2018) Ochsner J 18(1):98-100 +PMID: 29559880 [PubMed]
Zibis (2013) Eur Spine J 22(Suppl 3):S434-8 +PMID: 23179983 [PubMed]
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